Price range: $268.00 through $328.00
Background:
This gene encodes a protein that has been reported to function in signaling pathways governing transcriptional regulation and cell cycle progression. It may play a role in tumorigenesis and metastasis. A pseudogene of this gene is located on the long arm of chromosome 20. Alternative splicing results in multiple transcript variants that encode different isoforms. [provided by RefSeq, May 2013],
| Target | HSF-27 |
| Reactivity | Human, Mouse, Rat |
| Applications | WB, IHC, IF, ELISA |
| MW(Calculated) | |
| MW(Observed) | 60kD |
| Host Species | Rabbit |
| Isotype | IgG |
| Conjugate | |
| Modification | Unmodified |
| Recommended Dilution Ratio | WB 1:500-1:2000;IHC 1:100-1:300;ELISA 1:5000;IF 1:50-200 |
| Composition | Liquid in PBS containing 50% glycerol, 0.5% BSA and 0.02% sodium azide. |
| Source | |
| Purification Process | The antibody was affinity-purified from rabbit antiserum by affinity-chromatography using epitope-specific immunogen. |
| Purity | |
| Storage Condition | -15°C to -25°C/1 year(Do not lower than -25°C) |
| Concentration | 1 mg/ml |
| Clonality | Polyclonal |
| Clone Number | |
| Immunogen | The antiserum was produced against synthesized peptide derived from human CDK5RAP3. AA range:341-390 |
| Sequence | |
| Specificity | HSF-27 Polyclonal Antibody detects endogenous levels of HSF-27 protein. |
| Gene Name | CDK5RAP3 |
| Protein Name | CDK5 regulatory subunit-associated protein 3 |
| Alias | CDK5RAP3;IC53;MSTP016;OK/SW-cl.114;PP1553;CDK5 regulatory subunit-associated protein 3;CDK5 activator-binding protein C53;Protein HSF-27 |
| Organism-1 | Human |
| Gene ID-1 | 80279 |
| SwissProt-1 | Q96JB5 |
| Organism-2 | Mouse |
| Gene ID-2 | 80280 |
| SwissProt-2 | Q99LM2 |
| Organism-3 | Rat |
| Gene ID-3 | 80278 |
| SwissProt-3 | Q9JLH7 |
| Organism-4 | |
| Gene ID-4 | |
| SwissProt-4 | |
| Cellular Localization | Nucleus . Cytoplasm . Cytoplasm, cytoskeleton, microtubule organizing center, centrosome . Colocalizes and associates with microtubules. . |
